# 614373

AMYOTROPHIC LATERAL SCLEROSIS 16, JUVENILE; ALS16


Phenotype-Gene Relationships

Location Phenotype Phenotype
MIM number
Inheritance Phenotype
mapping key
Gene/Locus Gene/Locus
MIM number
9p13.3 ?Amyotrophic lateral sclerosis 16, juvenile 614373 AR 3 SIGMAR1 601978
Clinical Synopsis
 
Phenotypic Series
 

INHERITANCE
- Autosomal recessive
MUSCLE, SOFT TISSUES
- Limb muscle weakness, upper and lower
NEUROLOGIC
Central Nervous System
- Lower motor neuron signs
- Upper motor neuron signs
- Spasticity
- Hyperreflexia
- Muscle weakness, distal, upper and lower
- Neurophysiologic studies show evidence of denervation and renervation
- Enlarged motor unit action potentials
- Normal cognition
MISCELLANEOUS
- Age of onset 1 to 2 years
- Onset in lower limbs
- Progresses to involve upper limbs
- Slowly progressive
- Six patients from 1 Saudi Arabian family have been reported (last curated December 2011)
- Two of 6 patients became wheelchair-bound by age 20 years
MOLECULAR BASIS
- Caused by mutation in the sigma nonopioid intracellular receptor 1 gene (SIGMAR1, 601978.0001)
Amyotrophic lateral sclerosis - PS105400 - 35 Entries
Location Phenotype Inheritance Phenotype
mapping key
Phenotype
MIM number
Gene/Locus Gene/Locus
MIM number
1p36.22 Frontotemporal lobar degeneration, TARDBP-related AD 3 612069 TARDBP 605078
1p36.22 Amyotrophic lateral sclerosis 10, with or without FTD AD 3 612069 TARDBP 605078
2p13.1 {Amyotrophic lateral sclerosis, susceptibility to} AR, AD 3 105400 DCTN1 601143
2q33.1 Amyotrophic lateral sclerosis 2, juvenile AR 3 205100 ALS2 606352
2q34 Amyotrophic lateral sclerosis 19 AD 3 615515 ERBB4 600543
2q35 Amyotrophic lateral sclerosis 22 with or without frontotemporal dementia AD 3 616208 TUBA4A 191110
3p11.2 Amyotrophic lateral sclerosis 17 AD 3 614696 CHMP2B 609512
4q33 {Amyotrophic lateral sclerosis, susceptibility to, 24} AD 3 617892 NEK1 604588
5q31.2 Amyotrophic lateral sclerosis 21 AD 3 606070 MATR3 164015
5q35.3 Frontotemporal dementia and/or amyotrophic lateral sclerosis 3 AD 3 616437 SQSTM1 601530
6q21 Amyotrophic lateral sclerosis 11 AD 3 612577 FIG4 609390
9p21.2 Frontotemporal dementia and/or amyotrophic lateral sclerosis 1 AD 3 105550 C9orf72 614260
9p13.3 ?Amyotrophic lateral sclerosis 16, juvenile AR 3 614373 SIGMAR1 601978
9p13.3 Amyotrophic lateral sclerosis 14, with or without frontotemporal dementia 3 613954 VCP 601023
9q34.13 Amyotrophic lateral sclerosis 4, juvenile AD 3 602433 SETX 608465
10p13 Amyotrophic lateral sclerosis 12 3 613435 OPTN 602432
10q22.3 Amytrophic lateral sclerosis 23 AD 3 617839 ANXA11 602572
12q13.12 {Amyotrophic lateral sclerosis, susceptibility to} AR, AD 3 105400 PRPH 170710
12q13.13 Amyotrophic lateral sclerosis 20 AD 3 615426 HNRNPA1 164017
12q13.3 {Amyotrophic lateral sclerosis, susceptibility to, 25} AD 3 617921 KIF5A 602821
12q14.2 Frontotemporal dementia and/or amyotrophic lateral sclerosis 4 AD 3 616439 TBK1 604834
12q24.12 Spinocerebellar ataxia 2 AD 3 183090 ATXN2 601517
12q24.12 {Amyotrophic lateral sclerosis, susceptibility to, 13} AD 3 183090 ATXN2 601517
14q11.2 Amyotrophic lateral sclerosis 9 3 611895 ANG 105850
15q21.1 Amyotrophic lateral sclerosis 5, juvenile AR 3 602099 SPG11 610844
16p11.2 Amyotrophic lateral sclerosis 6, with or without frontotemporal dementia 3 608030 FUS 137070
17p13.2 Amyotrophic lateral sclerosis 18 3 614808 PFN1 176610
18q21 Amyotrophic lateral sclerosis 3 AD 2 606640 ALS3 606640
20p13 Amyotrophic lateral sclerosis 7 2 608031 ALS7 608031
20q13.32 Amyotrophic lateral sclerosis 8 AD 3 608627 VAPB 605704
21q22.11 Amyotrophic lateral sclerosis 1 AR, AD 3 105400 SOD1 147450
22q11.23 Frontotemporal dementia and/or amyotrophic lateral sclerosis 2 AD 3 615911 CHCHD10 615903
22q12.2 ?{Amyotrophic lateral sclerosis, susceptibility to} AR, AD 3 105400 NEFH 162230
Xp11.21 Amyotrophic lateral sclerosis 15, with or without frontotemporal dementia XLD 3 300857 UBQLN2 300264
Not Mapped Amyotrophic lateral sclerosis, juvenile, with dementia 205200 ALSDC 205200

TEXT

A number sign (#) is used with this entry because of evidence that juvenile amyotrophic lateral sclerosis-16 (ALS16) is caused by homozygous mutation in the SIGMAR1 gene (601978) on chromosome 9p13. One such family has been reported.


Clinical Features

Al-Saif et al. (2011) reported a consanguineous Saudi Arabian family in which 6 individuals had early-childhood onset of a neurologic disorder consistent with juvenile ALS, according to the El Escorial criteria (Brooks, 1994). Lower limb spasticity with hyperreflexia and weakness were noted at the age of 1 to 2 years. By age 9 or 10, affected individuals showed weakness of the hand and forearm muscles, which progressed to paralysis of the forearm extensors and triceps. By the age of 20 years, 2 patients used wheelchairs. None of the patients had respiratory or bulbar muscle weakness, and sensory and cerebellar functions were normal. Neurophysiologic tests showed evidence of denervation and reinnervation in limb muscles; motor unit potentials were enlarged, polyphasic, and fast firing, with normal sensory nerve action potentials and somatosensory evoked potentials. Brain imaging did not reveal any abnormalities, and cognition was preserved.


Mapping

By homozygosity mapping of a consanguineous Saudi Arabian family with early-childhood onset of ALS, Al-Saif et al. (2011) found linkage to a 120-kb region on chromosome 9p13.3.


Molecular Genetics

By homozygosity mapping followed by candidate gene sequencing, Al-Saif et al. (2011) identified a homozygous pathogenic mutation in the SIGMAR1 gene (601978.0001) in affected members of a consanguineous Saudi Arabian family with early-childhood onset of ALS.


REFERENCES

  1. Al-Saif, A., Al-Mohanna, F., Bohlega, S. A mutation in sigma-1 receptor causes juvenile amyotrophic lateral sclerosis. Ann. Neurol. 70: 913-919, 2011. [PubMed: 21842496, related citations] [Full Text]

  2. Brooks, B. R. El Escorial World Federation of Neurology criteria for the diagnosis of amyotrophic lateral sclerosis. Subcommittee on Motor Neuron Diseases/Amyotrophic Lateral Sclerosis of the World Federation of Neurology Research Group on Neuromuscular Diseases and the El Escorial 'Clinical limits of amyotrophic lateral sclerosis' workshop contributors. J. Neurol. Sci. 124 Suppl.: 96-107, 1994. [PubMed: 7807156, related citations] [Full Text]


Creation Date:
Cassandra L. Kniffin : 12/5/2011
ckniffin : 08/03/2015
terry : 7/5/2012
carol : 12/8/2011
ckniffin : 12/8/2011

# 614373

AMYOTROPHIC LATERAL SCLEROSIS 16, JUVENILE; ALS16


ORPHA: 300605;   DO: 0060207;  


Phenotype-Gene Relationships

Location Phenotype Phenotype
MIM number
Inheritance Phenotype
mapping key
Gene/Locus Gene/Locus
MIM number
9p13.3 ?Amyotrophic lateral sclerosis 16, juvenile 614373 Autosomal recessive 3 SIGMAR1 601978

TEXT

A number sign (#) is used with this entry because of evidence that juvenile amyotrophic lateral sclerosis-16 (ALS16) is caused by homozygous mutation in the SIGMAR1 gene (601978) on chromosome 9p13. One such family has been reported.


Clinical Features

Al-Saif et al. (2011) reported a consanguineous Saudi Arabian family in which 6 individuals had early-childhood onset of a neurologic disorder consistent with juvenile ALS, according to the El Escorial criteria (Brooks, 1994). Lower limb spasticity with hyperreflexia and weakness were noted at the age of 1 to 2 years. By age 9 or 10, affected individuals showed weakness of the hand and forearm muscles, which progressed to paralysis of the forearm extensors and triceps. By the age of 20 years, 2 patients used wheelchairs. None of the patients had respiratory or bulbar muscle weakness, and sensory and cerebellar functions were normal. Neurophysiologic tests showed evidence of denervation and reinnervation in limb muscles; motor unit potentials were enlarged, polyphasic, and fast firing, with normal sensory nerve action potentials and somatosensory evoked potentials. Brain imaging did not reveal any abnormalities, and cognition was preserved.


Mapping

By homozygosity mapping of a consanguineous Saudi Arabian family with early-childhood onset of ALS, Al-Saif et al. (2011) found linkage to a 120-kb region on chromosome 9p13.3.


Molecular Genetics

By homozygosity mapping followed by candidate gene sequencing, Al-Saif et al. (2011) identified a homozygous pathogenic mutation in the SIGMAR1 gene (601978.0001) in affected members of a consanguineous Saudi Arabian family with early-childhood onset of ALS.


REFERENCES

  1. Al-Saif, A., Al-Mohanna, F., Bohlega, S. A mutation in sigma-1 receptor causes juvenile amyotrophic lateral sclerosis. Ann. Neurol. 70: 913-919, 2011. [PubMed: 21842496] [Full Text: https://doi.org/10.1002/ana.22534]

  2. Brooks, B. R. El Escorial World Federation of Neurology criteria for the diagnosis of amyotrophic lateral sclerosis. Subcommittee on Motor Neuron Diseases/Amyotrophic Lateral Sclerosis of the World Federation of Neurology Research Group on Neuromuscular Diseases and the El Escorial 'Clinical limits of amyotrophic lateral sclerosis' workshop contributors. J. Neurol. Sci. 124 Suppl.: 96-107, 1994. [PubMed: 7807156] [Full Text: https://linkinghub.elsevier.com/retrieve/pii/0022-510X(94)90191-0]


Creation Date:
Cassandra L. Kniffin : 12/5/2011
Edit History:
ckniffin : 08/03/2015
terry : 7/5/2012
carol : 12/8/2011
ckniffin : 12/8/2011