Treatment of systemic sclerosis
EULAR recommendations for the treatment of systemic sclerosis: 2023 update
Annals of the Rheumatic Diseases (ARD)
is a leading rheumatology journal from EULAR, publishing original research, reviews and recommendations
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Latest EULAR Recommendations
Involvement of patient research partners in rheumatology research
EULAR recommendations for the involvement of patient research partners in rheumatology research: 2023 update
Management of psoriatic arthritis with pharmacological therapies
EULAR recommendations for the management of psoriatic arthritis with pharmacological therapies: 2023 update
Imaging in diagnosis and management of CiAs in clinical practice
2023 EULAR recommendations on imaging in diagnosis and management of crystal-induced arthropathies in clinical practice
Authors
Annals of the Rheumatic Diseases will be published by Elsevier from January 2025. All new manuscript submissions should be submitted online at https://www.editorialmanager.com/ards/default2.aspx.
Latest Articles
Autoimmunity:
Systematic literature review and meta-analysis informing the EULAR points to consider on the initiation of targeted therapies in patients with inflammatory arthritis and a history of cancer20 December 2024
Recommendation:
2024 EULAR points to consider on the initiation of targeted therapies in patients with inflammatory arthritis and a history of cancer20 December 2024
Recommendation:
Expert consensus recommendations for the diagnosis and treatment of chronic non-bacterial osteitis (CNO) in adults28 November 2024
Recommendations
The EULAR Standing Committees regularly establish and publish recommendations for the treatment of various rheumatic disorders. Each of these recommendations is developed by a group of specialists in their field.
All EULAR recommendations are free to access.
Each recommendation also has an accompanying lay summary, which is also free to access.
Video Abstract
m6A RNA methylation controls salivary gland epithelial cell function and has a protective role in Sjögren’s disease
Conclusions: Impairment of m6A modifications in SGEC in response to inflammatory triggers favour the formation of dsRNA, potentially amplifying the interferon loop and contributing to SjD pathogenesis.
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